The idea of inducing hypothermia has many reasons and as to what the potential outcome may lead to. The NFL player was hypothermia induced (my case hypothermia happened naturally). Yes he walks and is having great recovery but there is much as to why, what, when, and how. The who aspect of it is of relevance, but may not be as much as thought.
NFL player in great physical shape and such, true, but that didnt grant him such a large edge in his recovery. True to the 'naked' eye, but to me, all complexities can be reduced to a simpler or more basic element when analyzed/viewed at logically (and of course sought hard enough). After all, that NFL player is functioning on a drastically greater level, than most SCI patients, but how or why? Why induce him with hypothermia? Based on the info I've researched on the brain, etc (along with other 'tools' that I did & use) I've come to know that in paralysis there's alot of inflammation around the injury site (especially regarding SCI). This inflammation poses problems for the patient's recovery.
"Hypothermia, it is notable that even a small drop in temperature encourages cell membrane stability during periods of oxygen deprivation. For this reason, a drop in body temperature helps prevent an influx of unwanted ions during an ischemic insult. By making the cell membrane more impermeable, hypothermia helps prevent the cascade of reactions set off by oxygen deprivation." (dq) Hence, the degree of paralysis is in relationship to the degree of permeation.
My further research tells me that in my case when my 'lungs collapsed' something else may have taken place especially when all various aspects of what occured are considered. The 'something else' may have been 'Transpulmonary Cooling.'
"Transpulmonary Cooling induces therapeutic hypothermia by using the lungs as the heat exchanging mechanism. The large surface area of the pulmonary alveoli is utilized to exchange heat from the body to inspired gases. In the lungs, blood comes in close proximity to the inspired gases, as they are separated by the alveolar membrane which is only a few microns in thickness. The gossamer thinness of the alveolar membrane and the large surface area (about 70-90 square meters) of the lungs are ideally suited for heat exchange." (dq)
And all of this ties back into the idea that suggests that axonal damage and inflammation are linked.
This information has been researched and can be found on the internet and so forth. I will cease here to return again with more, but I'm tired, sorry. To be continued.
"direct quotes" or (dq)
'my quotes'
